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Results for "

NMN

" in MedChemExpress (MCE) Product Catalog:

By Targets:	Endogenous Metabolite (3) NAMPT (1) Toll-like Receptor (TLR) (1)
By Research Areas:	Cancer (3) Metabolic Disease (1) Neurological Disease (3)

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Cat. No.	Product Name	Target	Research Areas	Chemical Structure

HY-F0004

β-Nicotinamide mononucleotide 10+ Cited Publications β-NM; NMN	Endogenous Metabolite	Neurological Disease Cancer
β-nicotinamide mononucleotide (β-NM) is a product of the nicotinamide phosphoribosyltransferase (NAMPT) reaction and a key NAD ⁺ intermediate. The pharmacological activities of β-nicotinamide mononucleotide include its role in cellular biochemical functions, cardioprotection, diabetes, Alzheimer's disease, and complications associated with obesity .

HY-W615108B

β-Nicotinamide mononucleotide, reduced form disodium NMNH, reduced form disodium	Endogenous Metabolite	Cancer
β-Nicotinamide mononucleotide, reduced form disodium (β-NMN) is the oxidized form of NAD+ precursor and is a NAD+ enhancer. β-Nicotinamide mononucleotide, reduced form disodium can be reduced to dihydronicotinamide mononucleotide (NMNH). NMNH inhibits glycolysis, TCA cycle, and cell growth .

HY-F0004S

β-Nicotinamide mononucleotide-d4 β-NM-d₄; NMN-d₄	Endogenous Metabolite	Neurological Disease Cancer
β-Nicotinamide mononucleotide-d₄ is the deuterium labeled β-Nicotinamide mononucleotide. β-nicotinamide mononucleotide (β-NM) is a product of the nicotinamide phosphoribosyltransferase (NAMPT) reaction and a key NAD+ intermediate. The pharmacological activities of β-nicotinamide mononucleotide include its role in cellular biochemical functions, cardioprotection, diabetes, Alzheimer's disease, and complications associated with obesity[1].

HY-129522

Sulfo-ara-F-NMN 2 Publications Verification CZ-48	Toll-like Receptor (TLR)	Neurological Disease
Sulfo-ara-F-NMN (CZ-48) is a mimetic of nicotinamide mononucleotide (NMN). Sulfo-ara-F-NMN acts selectively, activating SARM1 but inhibiting CD38 (IC₅₀ around 10 μM). Sulfo-ara-F-NMN induces intracellular cyclic ADP-ribose (cADPR) production .

HY-126255

SBI-797812	NAMPT	Metabolic Disease
SBI-797812 is an orally active nicotinamide phosphoribosyltransferase (NAMPT) activator. SBI-797812 shifts NAMPT to NMN formation, increases NAMPT affinity for ATP, stabilizes phosphorylated NAMPT, promotes consumption of the pyrophosphate by-product, and blunts feedback inhibition by NAD ⁺. SBI-797812 increases intracellular nicotinamide mononucleotide (NMN) and elevates liver NAD ⁺ in mice .

Cat. No.	Product Name	Category	Target	Chemical Structure

HY-F0004

β-Nicotinamide mononucleotide 10+ Cited Publications β-NM; NMN	Structural Classification Other disease Neurological Disease Classification of Application Fields Anti-aging Source classification Endogenous metabolite Human Gut Microbiota Metabolites Microorganisms Ketones, Aldehydes, Acids Disease markers Disease Research Fields Cancer	Endogenous Metabolite
β-nicotinamide mononucleotide (β-NM) is a product of the nicotinamide phosphoribosyltransferase (NAMPT) reaction and a key NAD ⁺ intermediate. The pharmacological activities of β-nicotinamide mononucleotide include its role in cellular biochemical functions, cardioprotection, diabetes, Alzheimer's disease, and complications associated with obesity .

HY-W615108B

β-Nicotinamide mononucleotide, reduced form disodium NMNH, reduced form disodium	Structural Classification Natural Products Classification of Application Fields Source classification Endogenous metabolite Disease Research Fields Cancer	Endogenous Metabolite
β-Nicotinamide mononucleotide, reduced form disodium (β-NMN) is the oxidized form of NAD+ precursor and is a NAD+ enhancer. β-Nicotinamide mononucleotide, reduced form disodium can be reduced to dihydronicotinamide mononucleotide (NMNH). NMNH inhibits glycolysis, TCA cycle, and cell growth .

Species:	Human (1) Mouse (1) Cynomolgus (1)
Tag:	His (1) Fc (2)
Source:	HEK293 (3)

Cat. No.	Compare	Product Name	Species	Source

HY-P73743

	Neurotensin Protein, Human (HEK293, Fc) Neurotensin; neuromedin N; NMN-125; NMN; NTS	Human	HEK293
	Neurotensin, a neuropeptide, potentially modulates fat metabolism through endocrine or paracrine mechanisms, inducing smooth muscle contraction. It interacts with specific receptors like NTSR1 and binds to proteins, including SORT1 and SORL1, indicating involvement in diverse cellular processes and potential regulatory functions in fat metabolism. Neurotensin Protein, Human (HEK293, Fc) is the recombinant human-derived Neurotensin protein, expressed by HEK293 , with C-hFc labeled tag. The total length of Neurotensin Protein, Human (HEK293, Fc) is 148 a.a., with molecular weight of ~41 kDa.

HY-P77817

	NTS1/NTSR1 Protein, Mouse (HEK293, Fc) NT; NTR; NTR1; NTRH; NTRR; NTS1; NTSR1; NMN-125; NN; NT/N	Mouse	HEK293
	NTS1/NTSR1, the neurotensin receptor 1, potentially regulates fat metabolism through endocrine or paracrine mechanisms. Interacting with its ligand, neurotensin, NTS1/NTSR1 induces smooth muscle contraction. The receptor's interactions with neurotensin and other proteins like SORT1 and SORL1 suggest involvement in diverse cellular processes, indicating potential implications for fat metabolism regulation. NTS1/NTSR1 Protein, Mouse (HEK293, Fc) is the recombinant mouse-derived NTS1/NTSR1 protein, expressed by HEK293 , with C-hFc labeled tag. The total length of NTS1/NTSR1 Protein, Mouse (HEK293, Fc) is 140 a.a., with molecular weight of 48-53 kDa.

HY-P701029

	NTS1/NTSR1 Protein, Cynomolgus (HEK293, His) NT; NTR; NTR1; NTRH; NTRR; NTS1; NTSR1; NMN-125; NN; NT/N; NTS	Cynomolgus	HEK293
	NTS1/NTSR1 protein, as the receptor for neurotensin, regulates fat metabolism through potential endocrine and paracrine functions. The neuropeptide exerts influence on smooth muscle contraction. NTS1/NTSR1 Protein, Cynomolgus (HEK293, His) is the recombinant cynomolgus-derived NTS1/NTSR1 protein, expressed by HEK293 , with C-His labeled tag. The total length of NTS1/NTSR1 Protein, Cynomolgus (HEK293, His) is 140 a.a., with molecular weight of 20-25 kDa.

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Cat. No.	Product Name	Chemical Structure

HY-F0004S

β-Nicotinamide mononucleotide-d4
β-Nicotinamide mononucleotide-d₄ is the deuterium labeled β-Nicotinamide mononucleotide. β-nicotinamide mononucleotide (β-NM) is a product of the nicotinamide phosphoribosyltransferase (NAMPT) reaction and a key NAD+ intermediate. The pharmacological activities of β-nicotinamide mononucleotide include its role in cellular biochemical functions, cardioprotection, diabetes, Alzheimer's disease, and complications associated with obesity[1].

β-Nicotinamide mononucleotide-d4

β-Nicotinamide mononucleotide-d₄ is the deuterium labeled β-Nicotinamide mononucleotide. β-nicotinamide mononucleotide (β-NM) is a product of the nicotinamide phosphoribosyltransferase (NAMPT) reaction and a key NAD+ intermediate. The pharmacological activities of β-nicotinamide mononucleotide include its role in cellular biochemical functions, cardioprotection, diabetes, Alzheimer's disease, and complications associated with obesity[1].

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