2. Natural Products
  3. Disease Research Fields Plants Terpenoids
  4. Araliaceae Cardiovascular Disease Triterpenes
  5. Panax notoginseng (Burkill) F. H. Chen ex C. H. Chow
  6. Notoginsenoside R3

Notoginsenoside R3 is an active principle components of P. notoginseng. Notoginsenoside R3 act as a signaling molecule that is associated with various physiological effects. P. notoginseng and its components are used as traditional Chinese medicine for cardiovascular disease.

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Notoginsenoside R3 Chemical Structure

Notoginsenoside R3 Chemical Structure

CAS No. : 87741-76-2

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Based on 1 publication(s) in Google Scholar

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Description

Notoginsenoside R3 is an active principle components of P. notoginseng. Notoginsenoside R3 act as a signaling molecule that is associated with various physiological effects. P. notoginseng and its components are used as traditional Chinese medicine for cardiovascular disease[1].

Molecular Weight

963.15

Formula

C48H82O19
CAS No.
Appearance

Solid

Color

White to off-white

SMILES

C[C@]12[C@@](C[C@H]([C@@]3([H])[C@]2(CC[C@]3([H])[C@@](CC/C=C(C)/C)(C)O[C@@H]4O[C@@H]([C@H]([C@@H]([C@H]4O)O)O)CO[C@@H]5O[C@@H]([C@H]([C@@H]([C@H]5O)O)O)CO)C)O)([H])[C@@]6([C@@](C(C)([C@H](CC6)O)C)([H])[C@H](C1)O[C@@H]7O[C@@H]([C@H]([C@@H]([C@H]7O)O)O)CO)C
Structure Classification
Initial Source
Shipping

Room temperature in continental US; may vary elsewhere.
Storage
Powder -20°C 3 years
4°C 2 years
In solvent -80°C 6 months
-20°C 1 month
Purity & Documentation
References
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Notoginsenoside R3 Related Classifications

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Help & FAQs
  • Do most proteins show cross-species activity?

    Species cross-reactivity must be investigated individually for each product. Many human cytokines will produce a nice response in mouse cell lines, and many mouse proteins will show activity on human cells. Other proteins may have a lower specific activity when used in the opposite species.

Keywords:

Notoginsenoside R387741-76-2Notoginsenoside R 3Notoginsenoside R-3OthersP. notoginsengphysiologicalChinese medicinecardiovascular diseaseInhibitorinhibitorinhibit

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