2. Anti-infection Metabolic Enzyme/Protease
  3. Parasite Endogenous Metabolite
  4. 19,20-Epoxycytochalasin D

19,20-Epoxycytochalasin D, a cytochalasin, is a fungal metabolite from Nemania sp. 19,20-Epoxycytochalasin D shows potent in vitro antiplasmodial activity and phytotoxicity.

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19,20-Epoxycytochalasin D Chemical Structure

19,20-Epoxycytochalasin D Chemical Structure

CAS No. : 191349-10-7

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Based on 1 publication(s) in Google Scholar

19,20-Epoxycytochalasin D Related Antibodies

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Description

19,20-Epoxycytochalasin D, a cytochalasin, is a fungal metabolite from Nemania sp. 19,20-Epoxycytochalasin D shows potent in vitro antiplasmodial activity and phytotoxicity[1].

IC50 & Target

Plasmodium

 

Microbial Metabolite

 

In Vitro

19,20-Epoxycytochalasin D shows moderate toxicity to BT-549 and LLC-PK11 cell lines, with IC50 values of 7.84 μM and 8.4 μM, respectively[1].

MedChemExpress (MCE) has not independently confirmed the accuracy of these methods. They are for reference only.

19,20-Epoxycytochalasin D Related Antibodies
Molecular Weight

523.62

Formula

C30H37NO7
CAS No.
SMILES

CC(O[C@H]([C@@]1([H])[C@@]([C@@](O)(C2=O)C)([H])O1)[C@@]([C@](/C=C/C[C@@H]2C)([H])[C@@H]3O)(C(N4)=O)[C@]([C@@H](C3=C)C)([H])[C@@H]4CC5=CC=CC=C5)=O
Structure Classification
Initial Source

Xylaria hypoxylon
Shipping

Room temperature in continental US; may vary elsewhere.
Storage

Please store the product under the recommended conditions in the Certificate of Analysis.
Purity & Documentation
References
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19,20-Epoxycytochalasin D Related Classifications

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  • Do most proteins show cross-species activity?

    Species cross-reactivity must be investigated individually for each product. Many human cytokines will produce a nice response in mouse cell lines, and many mouse proteins will show activity on human cells. Other proteins may have a lower specific activity when used in the opposite species.

Keywords:

19,20-Epoxycytochalasin D191349-10-7ParasiteEndogenous Metaboliteantiplasmodial activityfungal metabolitephytotoxicityInhibitorinhibitorinhibit

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