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Results for "

EC 3.1.1.7

" in MedChemExpress (MCE) Product Catalog:

10

Inhibitors & Agonists

2

Recombinant Proteins

8

Oligonucleotides

Cat. No. Product Name Target Research Areas Chemical Structure
  • HY-P2860
    Acetylcholinesterase, Fly head

    ACHE; EC 3.1.1.7

    		 Cholinesterase (ChE) Neurological Disease
    Acetylcholinesterase, Fly head (ACHE; EC 3.1.1.7) is a cholinergic enzyme mainly found in neuromuscular junctions and cholinergic type chemical synapses used in biochemical research. Acetylcholinesterase, Fly head catalyzes the breakdown or hydrolysis of acetylcholine and some other choline esters that act as neurotransmitters into acetate and choline. Acetylcholinesterase, Fly head's main role is to terminate neuronal transmission and signaling between synapses to prevent ACh spread and activation of nearby receptors .
    Acetylcholinesterase, Fly head
  • HY-15228
    RX-3117
    1 Publications Verification

    TV-1360; Fluorocyclopentenylcytosine

    		 Nucleoside Antimetabolite/Analog Apoptosis Cancer
    RX-3117 (TV-1360) is a potent and orally active anticancer and antimetaboliteagent. RX-3117 inhibits DNA methyltransferase 1 (DNMT1). RX-3117 shows antiproliferative and anti-tumour activity. RX-3117 induces cell cycle arrest at S phase and apoptosis .
    RX-3117
  • HY-R00547
    hsa-miR-3117-3p mimic

    		MicroRNA Cancer
    hsa-miR-3117-3p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    hsa-miR-3117-3p mimic
    hsa-miR-3117-3p mimic
  • HY-R00548
    hsa-miR-3117-5p mimic

    		MicroRNA Cancer
    hsa-miR-3117-5p mimics are small, chemically synthesized double-stranded RNAs that mimic endogenous miRNAs and enable miRNA functional analysis by up-regulation of miRNA activity.
    hsa-miR-3117-5p mimic
    hsa-miR-3117-5p mimic
  • HY-RI00547
    hsa-miR-3117-3p inhibitor

    		MicroRNA Cancer
    hsa-miR-3117-3p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    hsa-miR-3117-3p inhibitor
    hsa-miR-3117-3p inhibitor
  • HY-RI00548
    hsa-miR-3117-5p inhibitor

    		MicroRNA Cancer
    hsa-miR-3117-5p inhibitors are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA inhibitors have full-length nucleotide 2'-methoxy modification. The miRNA inhibitors strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning.
    hsa-miR-3117-5p inhibitor
    hsa-miR-3117-5p inhibitor
  • HY-R00547A
    hsa-miR-3117-3p agomir

    		MicroRNA Cancer
    hsa-miR-3117-3p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-3117-3p agomir
    hsa-miR-3117-3p agomir
  • HY-R00548A
    hsa-miR-3117-5p agomir

    		MicroRNA Cancer
    hsa-miR-3117-5p agomirs are chemically-modified double-strand miRNA mimics with modified mature miRNA strand: 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 3' end cholesterol group, and full-length nucleotide 2'-methoxy modification. They are designed to mimic endogenous miRNAs and recommended for miRNA functional studies. Compared with miRNA mimics, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-3117-5p agomir
    hsa-miR-3117-5p agomir
  • HY-RI00547A
    hsa-miR-3117-3p antagomir

    		MicroRNA Cancer
    hsa-miR-3117-3p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-3117-3p antagomir
    hsa-miR-3117-3p antagomir
  • HY-RI00548A
    hsa-miR-3117-5p antagomir

    		MicroRNA Cancer
    hsa-miR-3117-5p antagomirs are chemically-modified oligonucleotides that hybridize with mature miRNAs. The miRNA antagomirs have 2 phosphorothioates at the 5' end, 4 phosphorothioates at the 3' end, 1 cholesterol group at the 3' end, and full-length nucleotide 2'-methoxy modification. The miRNA antagomirs strongly compete with mature miRNAs to prevent the complementary pairing of miRNAs and their target genes, thereby inhibiting miRNAs from functioning. Stability of miRNA antagomirs appears to be significantly higher than miRNA inhibitors, they exhibits enhanced cellular uptake, stability and regulatory activity in vivo.
    hsa-miR-3117-5p antagomir
    hsa-miR-3117-5p antagomir

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